A postdoctral fellow to study spinal cord injury

Location
White Plains in NY
Salary
standard salary for a postdoctral fellow
Posted
Jun 01, 2021
Closes
Jun 30, 2021
Position Type
Full Time

The Neural Connectivity Development in Physiology and Disease Laboratory at the Burke Neurological Institute /Weill Cornell Medicine, led by Yutaka Yoshida, Ph.D., is currently seeking Postdoctoral Fellows with laboratory experience in neuroscience. One of our main interests is to promote regeneration and motor recovery after spinal cord injury. Any postdoctoral fellow with neuroscience experience is welcome to apply for this position to study spinal cord injury.

https://burke.weill.cornell.edu/yoshida-lab

References)

  1. Gu Z., Sarrad N., Ueno M., Liang M., Li J., Enquist L.W., Baccei M.R., Martin J.H., and Yoshida Y. (2017). Skilled movements require non-apoptotic Bax/Bak pathway-mediated corticospinal circuit reorganization. Neuron, 94, 626-641.
  2. Gu Z., Kalamboglas J., Yoshioka S., Han W., Zhuo L., Imamura Kawasawa Y., Pochareddy S., LiZ.,  Liu F., Xu X., Wijeratne S., Ueno M., Blatz E., Salomone J., Kumanogoh A., Rasin M.R., Gebelein B., Weirauch M.T., Sestan N., Martin J.H., and Yoshida Y. (2017). Control of species-dependent cortico-motoneuronal connections underlying manual dexterity, Science, 357, 400-404.
  3. Ueno M., Ueno-Nakamura, Y., Li J, Gu Z., Niehaus J., Maezawa M., Crone S.A., Goulding M., Baccei M.L., and Yoshida Y. (2018). Corticospinal circuits from the sensory and motor cortex differentially regulate skilled movements through distinct interneurons. Cell Reports, 23, 1286-1300.
  4. Ueno M., Ueno-Nakamura Y., Niehaus J., Popovich P.G., and Yoshida Y. (2016). Silencing spinal interneurons inhibits immune suppressive autonomic reflexes caused by spinal cord injury. Nature Neuroscience, 19, 784-787
  5. Ueno M., Nakamura Y., Nakagawa H., Niehaus J., Maezawa M., Gu Z., Kumanogoh A., Takebayashi H., Lu Q.R., Takada M., and Yoshida Y. (2020). Olig2 induces semaphorin expression after spinal cord injury to limit axonal growth of injured neurons. Cerebral Cortex, 30, 5702-5716.

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