The Morrow Lab seeks a motivated postdoctoral research associate/fellow with experience in cell biology and a strong publication record.
This position will focus on molecular neuroscience with relevance to autism, schizophrenia, and/or neurologic disease in the areas of (1) endosome biology and/or (2) mitochondria and metabolism. Representative publications for projects include: Neuron 80(1):97-112, 2013 or PNAS 113(38): E5598-607, 2016.
Candidates with a strong training in molecular and cell biology, biochemistry, genetics, and/or neuroscience are desired. Also desired are skills in: mouse models; cell culture, including primary neuronal culture; and livecell imaging. Abilities in CRISPR-Cas9-based genome editing or metabolomics/metabolism are a plus.
The Morrow Lab is housed in the collaborative and multi-disciplinary Institute for Brain Science and the Department of Molecular Biology, Cell Biology and Biochemistry at Brown University in Providence, Rhode Island. The focus of Dr. Morrow’s research is normal mechanisms that regulate brain development, and genetic and cellular mechanisms that lead to neurodevelopmental and neuropsychiatric disorders. Using approaches that include mouse models and patient-derived iPSCs, Dr. Morrow’s research integrates basic research, translational research, and patient-oriented studies in the areas of human neurodevelopmental disorders, genetics/genomics, and mechanisms in cell biology.
Interested candidates should send a letter of application, current curriculum vitae, and names and contact information for three references to Eric M. Morrow MD PhD at: email@example.com.
Relevant lab webpage links:
van Dyck, L.I. and Morrow, E.M. (2017). Genetic control of postnatal human brain growth. Curr Opin Neurol 30: 114-124 (https://www.ncbi.nlm.nih.gov/pubmed/27898583)
Young-Pearse, T.L. and Morrow, E.M. (2016). Modeling developmental neuropsychiatric disorders with iPSC technology: Challenges and opportunities. Curr Opin Neurobiol 36: 66-73 (https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4738093/)
Ouyang, Q., Nakayama, T., Baytas, O., Davidson, S.M., Yang, C., Schmidt, M., Lizarraga, S.B., Mishra, S., El-Quessny, M., Niaz, S., Gul Butt, M., Imran Murtaza, S., Javed, A., Chaudhry, H.R., Vaughan, D.J., Hill, R.S., Partlow, J.N., Yoo, S.-Y., Lam, A.-T.N., Nasir, R., Al-Saffar, M., Barkovich, A.J., Schwede, M., Nagpal, S., Rajab, A., DeBerardinis, R.J., Housman, D.E., Mochida, G.H., and Morrow, E.M. (2016). Mutations in mitochondrial enzyme GPT2 cause metabolic dysfunction and neurological disease with developmental and progressive features. Proc Natl Acad Sci USA 113: E5598-E5607 (http://www.pnas.org/content/113/38/E5598.long).
Ouyang, Q., Lizarraga, S.B., Schmidt, M., Yang, U., Gong, J., Ellisor, D., Kauer, J.A., and Morrow, E.M. (2013). Christianson syndrome protein NHE6 modulates TrkB endosomal signaling required for neuronal circuit development. Neuron 80: 97-112 (http://www.cell.com/neuron/pdf/S0896-6273(13)00667-3.pdf).