We have an opening for a postdoctoral or Ph.D. position at the Rosenblum lab, Sagol Dept. of Neurobiology, University of Haifa, to study the role of eEF2 in learning and memory and in psychiatric disorders, using advanced methods of molecular biology, imaging, electrophysiology, and behavioral techniques. For additional information see http://neuroethocms.haifa.ac.il/rosenblum/
Candidates should hold a PhD or MSc degree. Knowledge in neurobiology and experience in one or more of the following fields are an advantage: advanced molecular biology, fluorescence-based microscopy techniques and image analysis, behavioral assays, stereotaxic administration of drugs and viral vectors, electrophysiological techniques, and cell culture. To apply, please send a CV, grade transcripts of B.A./B.Sc., M.A./M.Sc, and Ph.D. (including thesis scores where applicable), a short description of research interests, and the names and contact details of three academic referees to firstname.lastname@example.org
The laboratory for research of molecular and cellular mechanisms underlying learning and memory
The laboratory for research of molecular and cellular mechanisms underlying learning and memory, directed by Prof. Kobi Rosenblum, is affiliated to the Department of Neurobiology and Ethology, Haifa University, Israel. Our research aim is to understand how simple sensory information is encoded, conso...lidated and maintained in the cortex and elucidate the biological mechanisms underlying memory formation and consolidation in the mammalian brain.
Since our research is inherently multi-disciplinary, we combine research approaches from the field of biology, as well as psychology, integrating data ranging from the molecular to whole animal level. In our laboratory we employ state of the art equipment (e.g., confocal microscope, real-time PCR, electrophysiology, biochemistry and molecular biology), as well as a broad spectrum of techniques both in-vitro and in-vivo for the study of normal and abnormal cognitive function in behaving animals. Following recent results regarding the role of translation regulation in normal learning processes, we test the hypothesis that malfunction of similar biochemical pathways underlies cognitive dysfunction and sporadic Alzheimer's disease.